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analysis version v0.4.1evidence current through 2026-08-10as of 2026-08-11last reviewed 2026-08-11review status: publishedsite mode: publicationpatient approval: obtainedclinician review: not performedindexing: enabled
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Literature card · launch set

lit-0339

Fracture risk among patients with urolithiasis: a population-based cohort study

Melton LJ 3rd, Crowson CS, Khosla S, Wilson DM, O'Fallon WM (1998). Kidney International.

study design: cohortaccess: abstract-onlylicense: unknownrelevance: mediumBibliographic identity verified (identifier match only)

Study design and applicability are separate. A rigorous design in an unmatched population is not “strong evidence” for this record.

Bibliographic identity (not medical verification)

Status: Bibliographic identity verified (identifier match only). Identifier resolution proves bibliographic identity only; it does not prove that the card summary supports any medical claim, and it is not clinician review. Semantic verification count remains 0 for the catalog.

Applicability / quality notes

Filed in v0.4.0. Population cohort reporting that excess fracture risk in stone disease was confined to the spine, with no increase at hip, pelvis, humerus or forearm. Cited as a limit on how much a stone-and-calcium mechanism can explain when low readings appear at several sites at once. Applicability here is weak in a specific and stated way - this is a cohort of symptomatic stone formers, and the documented history is a single stone in one episode, so the comparison is to the mildest end of that cohort. Do not read it as evidence that stones are harmless to bone; the vertebral signal it reports is real.

Record overlap notes (research). one lifetime stone documented; low BMD at spine, hip and forearm

DOI: 10.1046/j.1523-1755.1998.00779.x · PMID: 9461107 · PMCID: —

Open external bibliographic record

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What cites this on the site

  • Hypothesis H3 (contradicting)

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